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blue tablet, small, R on one side, 031 reverse

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  • blue tablet, small, R on one side, 031 reverse

    Identify

  • #2
    benzodiazepine. used to treat nervousness,"panic attacks"
    this is a milder form of valium. habit forming.

    hope this helps. ironchef.

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    • #3
      Generic Name Alprazolam Tab 1 MG
      Imprint Code 031 / a logo -> R <-
      Color blue
      Shape round


      Kyle - Pharmacy Tech and Rockstar!

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      • #4
        That's right

        This pill is ALPRAZOLAM 1 MG
        a generic version of Xanax
        Imprint Code 031 / R (Purepac logo)
        Description blue, round, scored tablet

        Info at https://www.drugs.com/alprazolam.html

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        • #5
          RE: Small blue scored tablet with 031 and R on reverse side. Heres New Stuff

          Originally posted by ironchef View Post
          benzodiazepine. used to treat nervousness,"panic attacks"
          this is a milder form of valium. habit forming.

          hope this helps. ironchef.
          XANAX (generically alprazolam) 1 mg for sure, manufactured by Purepac if it is a small blue scored tablet with 031 on one side and the funny looking R on the reverse side. The only clarification. From a psychopharmacologic standpoint, Xanax is in a subclass called triazolobenzodiazepines, a term that Upjohn plausibly came up with in order toi distinguish their product over rival Valium (diazepam) and especially Ativan (lorazepam), it latest competitor at the time. One must NOT under any circumstances classify Xanax as a milder form of Valium because it simply is not true. It not only has a shorter half-life which means that a patient dependant on Xanax will have a more precipitous withdrawal and onset of withdrawal is quicker. Additionally, Xanax is classified as a "high potency" benzo (remember, be classy -its triazolobenzodiazepine) which means that it binds much tighter to the GABA receptors (thats how everythihng from Librium to Tranxene, Valium to Ativan work: by binding to the Gamma amino butyric acid receptor) and that leads to one feeling chilled out buzzed feeling. Valium has more profound muscle relaxant effects so you will see doctors use that in acutely anxious patients with lots of muscle tension. MY MESSAGE TO YOU ALL IS THIS: Xanax, when used in low doses for periods of time less than one month is perfectly warranted, with the duration of therapy being as short as possible. Xanax IS the most highly addictive of all the benzo's. Following the usual tapering schedule to get off Xanax of reducing the daily dose by ten percent every week is way too steep. I ask every one of you who has been on Xanax for more than 5 or 6 months and have tried to quit. IT is exceedingly rare for one to do it alone. A skilled substance abuse specialist who will "work with you" at your own pace and you feel comfortable with is key. I also challenge any physicians or pharmacists and review your records of just how many tims that you have seen a patient successfully wean off long term, moderate to high dose Xanax. Xanax is an excellent drug when used properly, but when it came out it was way overprescribed and there is an entire "culture" of Xanan and Ativan addicts out there and in most cases it is not their fault, they were hust following the advice of their physician. I've witnessed quite a bit of luck by bridging the patient to and equivalent dose of another longER acting high potency benzo called Klonopin (clonazepam). Some even try to switch from Xanax to an equipotent dose of Valium but sometimes it doesn't work and their are breakthrough withdrawal symptoms. Once the patient is stable on the equipotent dose of Klonopin, it may be titrated down, with high emphasis on involvement in a 12 step program or counselling. I am passionate about this topic and could not accept the false statement that
          Xanax is a milder Xanax. Maybe Xanax subjectively to the patient seems milder than Valium because they love the euphoric "kick" but aren't glued to the couch due to the moderate muscle relaxant effects. I welcome any and all feedback and am willing to try to answer any question. I am a newcomer as a result of reading the earlier post. I might next decide to write on appropriate tapering of Suboxone in opiate addiction,l another one of my pet peeves, because their manufacturer, like that of Xanax, does not make a tablet dose strenght low enough in order to get accurate microdoses when it is most important near the end of the drug taper and the patient actually gets off the med for good. I ripped this one out without editing. Please comment to me because I;m sure there is a lot of interest in this area.

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          • #6
            Excellent, very informative post! I for one very much look forward to reading future posts from you, I hope there's more to come.
            Cats

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            • #7
              You Are Most Welcome

              Originally posted by Cats Meow View Post
              Excellent, very informative post! I for one very much look forward to readifuture posts from you, I hope there's more to come.
              Cats
              Hello Cats Meow,

              Since that was my first post, you can imagine how cool that was when you left a remark so quickly. Go ahead, make my day, Ask me a question, I dare yoU

              Dave

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              • #8
                Okay Dave, it's funny, it's usually me answering the questions, not asking them! There is something maybe you can shed some light on, can you tell me what it is exactly that Buprenorphine and Methadone do to ravage pain receptors so viciously that makes even very slow taper and low mg drop offs to cold turkey so excruciatingly difficult and painful in the withdrawal process? I understand a long slow taper down to microdosing is crucial for success and partially reduces the w/d symptoms, but I'm more interested in what the difference is that it does at a say 1-2 mg drop, compared to other opioids, why is Bup and Meth so devastating to receptors, or is it more then just the receptors at fault?

                Please explain, I look forward to your academician response.
                Thanks again
                Cats

                Comment


                • #9
                  Along with Cat's question, I've been having a small debate with some co-workers. I can only imagine that the w/d off methadone is unbelievable. However, one co-worker tells me (not by experience) that once you're at 15-10mgs, it's all in your mind and you don't need to continue to taper. In essence, he says you should be able to stop cold turkey at 15mg. I don't agree with this, and personally this is when I see people have the most trouble. Who is "right"?

                  Comment


                  • #10
                    Med_Pharm_Wiz (Dave on the run)

                    Hi There,

                    I'll be able to give you an answer on that question Cats Meow, but I'm on the run and want to give proper attention to your inquiry. We ARE talking life/death decisions here you know and I don't want to mess you up. Please give me whatever history with timelines/dosages, and I'll get back to you with a customized taper which WILL work for you. Make no mistake, one way or another, you gotta pay the piper, but its NOT as bad as it sounds. TTYL. I don't know if personal emails are allowed, and I would provide mine if you think it may help.

                    Originally posted by Cats Meow View Post
                    Okay Dave, it's funny, it's usually me answering the questions, not asking them! There is something maybe you can shed some light on, can you tell me what it is exactly that Buprenorphine and Methadone do to ravage pain receptors so viciously that makes even very slow taper and low mg drop offs to cold turkey so excruciatingly difficult and painful in the withdrawal process? I understand a long slow taper down to microdosing is crucial for success and partially reduces the w/d symptoms, but I'm more interested in what the difference is that it does at a say 1-2 mg drop, compared to other opioids, why is Bup and Meth so devastating to receptors, or is it more then just the receptors at fault?

                    Please explain, I look forward to your academician response.
                    Thanks again
                    Cats

                    Comment


                    • #11
                      LOL, I don't take either drug, or any drug for that matter, my question is purely informational, for my own selfish interests.
                      Take your time, it's not exactly an easy question, a good answer is worth waiting for.
                      Thanks

                      Comment


                      • #12
                        The Co-Worker is WRONG

                        Originally posted by Asashi View Post
                        Along with Cat's question, I've been having a small debate with some co-workers. I can only imagine that the w/d off methadone is unbelievable. However, one co-worker tells me (not by experience) that once you're at 15-10mgs, it's all in your mind and you don't need to continue to taper. In essence, he says you should be able to stop cold turkey at 15mg. I don't agree with this, and personally this is when I see people have the most trouble. Who is "right"?
                        Dear Asashi,

                        Most people cannot just "jump off" I call it, at 10-15 mg/day without withdrawal symptoms that will eventually get them back to using. I see a lot of people who just switch over to and bomb themselves with muscle relaxant, benzodiazepines, alcohol, marijuanna, whatever it takes to snow themselves into enough of a stupor to not notice the withdrawal as much. I know one man who says he keeps 2 of the 8mg Suboxone handy so that if he needs to go to jail he can get off of his >>>>>> habit. BALONEY. If you are reading this CatsMeow, ur next, its almost 3 am and I'm exhausted.

                        Dave

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                        • #13
                          I think that is not Methadone. It's look like Xanax as i think.
                          Last edited by Anonymous; 08-19-2018, 10:47 PM.

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                          • #14
                            I'm with you on this one Cats Meow

                            Originally posted by John Weelsen View Post
                            I think that is not Methadone. It's look like Xanax as i think.
                            That is most definitely generic alprazolam 1.0 mg (ie., Xanax), the "triazolobenzodazepine." Its just a hopped up version of diazepam (Valium) or lorazepam (Ativan) and the pill itself does resemble a methadone 10 mg but is thicker and has a smaller diameter. Benzo's, and this includes Xanax are used to control ACUTE anxiety and some of them, like diazepam (Valium), have strong enough muscle relaxant properties that they can come in handy when formal muscle relaxants like methocarbamol (Robaxin), carisoprodol (SOMA), or cyclobenzaprine (Flexeril) are not available for backaches, sprains etcetera. I wouldn't want to find either of these in my daughter's purse. The 1 mg Xanax is a lot stronger buzz effect than a single methadone 10 mg, but the withdrawal from chronic Xanax use is very scary and dangerous as well. Whether Xanax or Methadone, and this gets into my response to Cats Meow: It all has to do with two important properties related to how many drugs work. One property is how strongly they bind to the receptor. Benzodiazepines bind to and render effects at the GABA receptor (gamm-amino butyric acid). Normally, chemicals in your body bind to and stimulate GABA, Benzo's like Ativan, Valium, the "triazolo" Benzo Xanax and, yup, you may have guessed it alcohol get the binding to the GABA receptors part right, but instead of stimulating it, they effectively block and protect the receptor from your own body's stimulatory chemicals. NET RESULT: CNS depression, anxiolysis amd there you have it with the Benzodiazepines effects. With methadone and Suboxone, the target receptor is the Opioid receptors in the brain. Normally, the brain's own opioids, endorphins and enkephalins, bind to these receptors and they have some effect in regulating mood. >>>>>> and vicodin bind to these and cause the buzz and euphoria. Methadone and Suboxone both bind to opioid receptors strongly, more strongly than the recreational drugs themselves. The other property drugs have at receptors is how potent their stimulatory or inhibitory effects are. Well you may have guessed, methdone and Suboxone actually bind to and displace narcotic opioids and that has something todo with why you get some nasty effects until you have found a stable dose and subsequent desired blood blood level. There is a difference between how methadone and Suboxone work. If anyone is interested, give me a holler, because I got to get up and get going with my day. Hope you all have a great weekend.
                            Dave

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                            • #15
                              Thanks Dave, but I already knew all of that. I was hoping to get more into the chemistry and bio-mechanics of the receptor vis-à-vis Bup and Meth. Thanks for trying, I realize this may not be your forte'.
                              Cats

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